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(62%) were serodiagnosed as HHV-6 infection.
Sixteen strains of HHV-6 from 31 patients (52%) whose
antibody titers had converted during convalescence were
isolated during the acute phase. The disease occurred
in infants from 3 months to 1 year of age and most frequently
at age 4-6 months. Antibody only to HHV-6 converted
in 23 of 50 patients (46%), and seroconversion to HHV-6
and dengue virus was observed in 7 patients (14%), and
to HHV-6 and Coxsackie B virus in 1 case (2%). In the
23 patients in whom seroconversion only to HHV-6 was
observed, all had fever and rash which appeared after
subsidence of the fever. Lymphadenopathy and relative
lymphocytosis were recognized, associated with diarrhea,
vomiting, running nose, cough and hepatomegaly. Febrile
convulsions were seen in some cases. All patients recovered
completely within a week.
Balachandra, K., K. Chimabutra, et
al. (1994). "High rate of reactivation of human
herpesvirus 6 in children with dengue hemorrhagic fever."
J.Infect.Dis. 170: 746-748.
K Balachandra, Osaka Univ, Microbial Dis Res Inst, Dept
Virol, Suita, Osaka 565, Japan
Bancroft, W. H., J. M. McCown, et
al. (1979). "Identification of dengue viruses from
the Caribbean by plaque-reduction neutralization test."
PAHO.Sci.Pub. 375: 173-178.
AB: A pool of whole-mouth saliva collected from five
human carriers of hepatitis B surface antigen, subtype
adr, was found to contain antigen particles with mean
diameters of 23.3 and 41.8 nm as seen by immune electron
microscopy. Two gibbons received subcutaneous injections
of the pooled saliva and developed serological and,
in at least one animal, biochemical evidence of hepatitis
B virus infection at 12 and 22 weeks, respectively.
Although none of eight other gibbons that were exposed
by the nasal or oral routes were infected, the experiment
demonstrated that human saliva can serve as a vehicle
for the transmission of hepatitis B virus
Bancroft, W. H., R. M. Scott, et al.
(1982). "Dengue-2 vaccine: infection of Aedes aegypti
mosquitoes by feeding on viremic recipients." Am.J.Trop.Med.Hyg.
31(6): 1229-1231.
Colonized Aedes aegypti mosquitoes were fed on voluntary
recipients of an experimental, live, attenuated, dengue
type 2 (PR-159/S-1) vaccine to estimate the frequency
of vector infection and the stability of the virus in
mosquitoes. Two volunteers were viremic at the time
of mosquito feeding, but only two of 114 mosquitoes
that took a viremic blood meal became infected with
the vaccine virus. Strains of virus recovered from the
bodies of the mosquitoes and the volunteer's blood retained
the temperature sensitivity and small plaque growth
characteristics of the vaccine virus. Dengue viral antigen
was not detectable in any of the mosquito heads by direct
immunofluorescence and in vitro virus transmission by
droplet feeding was not observed. This experiment showed
that vector mosqitoes can be infected with vaccine virus
by feeding on viremic vaccinees. Furthermore, the virus
is sufficiently stable to retain the in vitro growth
characteristics associated with the vaccine virus.
Bancroft, W. H., R. M. Scott, et al. (1984). "Dengue
virus type 2 vaccine: reactogenicity and immunogenicity
in soldiers." J.Infect.Dis. 149(6): 1005-1010.
 
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